Cl- has an unhydrated diameter of 190pm, K+=160, and Na+=100. Their hydrated diameters are 300, 300, and 450pm respectively. So it makes sense in a low ATP (unhydrated) why Cl- uses the same channel (https://en.m.wikipedia.org/wiki/Na-K-Cl_cotransporter). Really anything that has the same absolute charge and diameter should be able to go through the same channels. Which is why Ca channel blockers were found to stop Fe+2 accumulation.

On a side note, channels are interesting. Same as receptors. I’m not convinced they fully exist in the way we conventionally think about them. You would have to many local changes and is there enough surface area? All things I’m thinking about and researching.

I wonder if the trace minerals had something else like Se, I, Mn, etc that was also helping energy production. The Cl- ions shown to be slightly less inside the cell vs plasma (http://book.bionumbers.org/what-are-the-concentrations-of-different-ions-in-cells/) in a normal ATP state would definitely help with the membrane potential.

I guarantee people in the MgG don’t do well with the adrenal cocktails. They either don’t say anything and move onto another program, or do and are told to go slower, etc. Just like with Mg. I keep posting about B1 In there (focus on porkchops and removing inhibitors like sulfites because they’re against “synthetics”).